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- W2144908726 endingPage "321" @default.
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- W2144908726 abstract "I. Introduction IN 1953 lohn Morris, an obstetrician at Yale University, reported a series of 82 individuals (80 cases collated from the literature and two cases of his own) who had a female phenotype despite the presence of bilateral testes (1). Since his initial description, studies of the endocrinology, pathophysiology, biochemistry, and molecular biology of the androgen insensitivity syndrome (AIS) have provided insights into the role of androgens in male sex differentiation, the mechanisms of androgen action, and aspects of the structure/function relationships of the androgen receptor. AIS is an archetypal example of a hormone resistance disorder. Androgens are secreted by the testes of these 46,XY individuals in normal or increased amounts; however, due to defective androgen receptor (AR1) function, there is loss of target organ response to the hormone, and the effects of androgens are reduced or absent. Clinical disorders of the AR are reported far more commonly than resistance disorders of other m..." @default.
- W2144908726 created "2016-06-24" @default.
- W2144908726 creator A5011989818 @default.
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- W2144908726 creator A5079500025 @default.
- W2144908726 creator A5080189561 @default.
- W2144908726 creator A5091276012 @default.
- W2144908726 date "1995-06-01" @default.
- W2144908726 modified "2023-10-17" @default.
- W2144908726 title "Androgen Receptor Defects: Historical, Clinical, and Molecular Perspectives*" @default.
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