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• W2144992593 abstract "Abstract Aims Poor glycaemic control is associated with increased risk of microvascular disease in various organs including the eye and kidney, but the relationship between glycated haemoglobin (HbA 1c ) and microvascular function in skeletal muscle has not been described. We tested the association between HbA 1c and a measure of microvascular exchange capacity ( K f ) in skeletal muscle in people with central obesity at risk of developing Type 2 diabetes. Methods Microvascular function was measured in 28 women and 19 men [mean (± sd ) age 51 ± 9 years] with central obesity who did not have diabetes. We estimated insulin sensitivity by hyperinsulinaemic–euglycaemic clamp, visceral and total fatness by magnetic resonance imaging, fitness (VO 2 max by treadmill testing), physical activity energy expenditure [metabolic equivalents of tasks (METS) by use of the SenseWear Pro armband] and skeletal muscle microvascular exchange capacity ( K f ) by venous occlusion plethysmography. Results In regression modelling, age, sex and fasting plasma glucose accounted for 30.5% of the variance in HbA 1c ( r 2 = 0.31, P = 0.001). Adding K f to this model explained an additional 26.5% of the variance in HbA 1c ( r 2 = 0.57, P = 0.0001 and K f was strongly and independently associated with HbA 1c (standardized B coefficient −0.45 (95% confidence interval −0.19, −0.06), P = 0.001). Conclusions We found a strong negative independent association between a measure of skeletal muscle microvascular exchange capacity ( K f ) and HbA 1c . K f was associated with almost as much of the variance in HbA 1c as fasting plasma glucose." @default.
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• W2144992593 date "2009-11-01" @default.
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• W2144992593 title "Skeletal muscle microvascular exchange capacity is associated with hyperglycaemia in subjects with central obesity" @default.
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• W2144992593 doi "https://doi.org/10.1111/j.1464-5491.2009.02822.x" @default.
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