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- W2145110767 abstract "Platelets are a rich source of different chemokines and express chemokine receptors. CXCL4 is highly abundant in platelets and involved in promoting monocyte arrest from rolling and monocyte differentiation to macrophages. CXCL4 can also associate with CCL5 and amplify its effect on monocytes. The megakaryocyte CXCL7 gene product is proteolytically cleaved into the strong neutrophil chemoattractant, NAP-2, which has also been implicated in repair cell homing to vascular lesions. Platelet adhesion can induce release of CCL2 and CXCL8 from endothelial cells. Conversely, the chemokines CCL17, CCL22, and CXCL12 made by other cells amplify platelet activation. Platelet chemokines enhance recruitment of various hematopoietic cells to the vascular wall, fostering processes such as neointima formation, atherosclerosis, and thrombosis, but also vessel repair and regeneration after vascular injury." @default.
- W2145110767 created "2016-06-24" @default.
- W2145110767 creator A5020082525 @default.
- W2145110767 creator A5067336736 @default.
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- W2145110767 date "2008-11-01" @default.
- W2145110767 modified "2023-10-12" @default.
- W2145110767 title "Platelet Chemokines in Vascular Disease" @default.
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- W2145110767 doi "https://doi.org/10.1161/atvbaha.108.169417" @default.
- W2145110767 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/2657037" @default.
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