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- W2145832015 abstract "Albert, T. S. E., V. L. Tucker, and E. M. Renkin.Atrial natriuretic peptide levels and plasma volume contraction in acute alveolar hypoxia. J. Appl. Physiol. 82(1): 102–110, 1997.—Arterial oxygen tensions ([Formula: see text]), atrial natriuretic peptide (ANP) concentrations, and circulating plasma volumes (PV) were measured in anesthetized rats ventilated with room air or 15, 10, or 8% O 2 ( n = 5–7). After 10 min of ventilation, [Formula: see text] values were 80 ± 3, 46 ± 1, 32 ± 1, and 35 ± 1 Torr and plasma immunoreactive ANP (irANP) levels were 211 ± 29, 229 ± 28, 911 ± 205, and 4,374 ± 961 pg/ml, respectively. At[Formula: see text] ≤40 Torr, irANP responses were more closely related to inspired O 2 ( P = 0.014) than to[Formula: see text] ( P= 0.168). PV was 36.3 ± 0.5 μl/g in controls but 8.5 and 9.9% lower ( P ≤ 0.05) for 10 and 8% O 2 , respectively. Proportional increases in hematocrit were observed in animals with reduced PV; however, plasma protein concentrations were not different from control. Between 10 and 50 min of hypoxia, small increases (+40%) in irANP occurred in 15% O 2 ; however, there was no further change in PV, hematocrit, plasma protein, or irANP levels in the lower O 2 groups. Urine output tended to fall during hypoxia but was not significantly different among groups. These findings are compatible with a role for ANP in mediating PV contraction during acute alveolar hypoxia." @default.
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- W2145832015 date "1997-01-01" @default.
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- W2145832015 title "Atrial natriuretic peptide levels and plasma volume contraction in acute alveolar hypoxia" @default.
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- W2145832015 doi "https://doi.org/10.1152/jappl.1997.82.1.102" @default.
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