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- W2146364817 abstract "Clinical observations indicate that parkinsonians do not, or only exceptionally, exhibit epileptic phenomena and vice versa (Scholz, 1957; Klee, 1975). Yakovlev reported (1928) that when epileptic patients develop parkinsonism, the epilepsy pari passu disappears. The exact neurophysiological mechanisms underlying these pathological conditions are poorly understood. Yet, it is apparent, that both of these diseases dominantly involve the sensorimotor system. Thus the question appears to be of more than heuristic importance as to why these two disease states are mutu ally exclusive. Whereas epilepsy readily lends itself to experimental approaches in the domain of electrophysiology, parkinsonism does not, because only its morphological and biochemical features but not its clinical symptomatology can be reproduced in experimental animals. The normal integration of sensorimotor activities, and the electrophysiological consequences of caudatal and nigral damage (established sites of pathology in parkinsonism) to such integrative processes, have extensively been explored in felines and subhuman primates (Fvigyesi, 1975a, 1975b); these studies yielded data which, when extrapolated to parkinsonism, suggested the nature of elementary processes which underlie, at least some symptoms of, parkinsonism (Frigyesi, 1971; Frigyesi, et al., 1974)." @default.
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- W2146364817 date "1977-01-01" @default.
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- W2146364817 title "Parkinsonism and Epilepsy" @default.
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- W2146364817 doi "https://doi.org/10.1007/978-1-4684-2511-6_4" @default.
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