FRINK Linked Data Fragments server

Matches in SemOpenAlex for { <https://semopenalex.org/work/W2146827984> ?p ?o ?g. }

• W2146827984 endingPage "E29" @default.
• W2146827984 startingPage "E28" @default.
• W2146827984 abstract "Journal of Cardiovascular ElectrophysiologyVolume 24, Issue 12 p. E28-E29 Letter to the Editor Pacemaker Gene Mutations, Bradycardia, Arrhythmias and the Coupled Clock Theory YAEL YANIV Ph.D., Corresponding Author YAEL YANIV Ph.D. Laboratory of Cardiovascular Science Biomedical, Research Center Intramural Research Program, National Institute on Aging, NIH, Baltimore, Maryland, USAAddress for correspondence: Yael Yaniv, Ph.D., Laboratory of Cardiovascular Science, Biomedical Research Center, Intramural Research Program, National Institute on Aging, NIH, Baltimore, MD 21224-6825, USA. Fax: 410-558-8654; E-mail: [email protected]Search for more papers by this authorEDWARD G. LAKATTA M.D., EDWARD G. LAKATTA M.D. Laboratory of Cardiovascular Science Biomedical, Research Center Intramural Research Program, National Institute on Aging, NIH, Baltimore, Maryland, USASearch for more papers by this author YAEL YANIV Ph.D., Corresponding Author YAEL YANIV Ph.D. Laboratory of Cardiovascular Science Biomedical, Research Center Intramural Research Program, National Institute on Aging, NIH, Baltimore, Maryland, USAAddress for correspondence: Yael Yaniv, Ph.D., Laboratory of Cardiovascular Science, Biomedical Research Center, Intramural Research Program, National Institute on Aging, NIH, Baltimore, MD 21224-6825, USA. Fax: 410-558-8654; E-mail: [email protected]Search for more papers by this authorEDWARD G. LAKATTA M.D., EDWARD G. LAKATTA M.D. Laboratory of Cardiovascular Science Biomedical, Research Center Intramural Research Program, National Institute on Aging, NIH, Baltimore, Maryland, USASearch for more papers by this author First published: 17 July 2013 https://doi.org/10.1111/jce.12236Citations: 6Read the full textAboutPDF ToolsRequest permissionExport citationAdd to favoritesTrack citation ShareShare Give accessShare full text accessShare full-text accessPlease review our Terms and Conditions of Use and check box below to share full-text version of article.I have read and accept the Wiley Online Library Terms and Conditions of UseShareable LinkUse the link below to share a full-text version of this article with your friends and colleagues. Learn more.Copy URL No abstract is available for this article. References 1Nawathe PA, Yelena K, Oren RV, Milanesi R, Clancy CE, Lu JT, Moss AJ, Difrancesco D, Robinson RB: An LQTS6 MiRP1 mutation suppresses pacemaker current and is associated with sinus bradycardia. J Cardiovasc Electrophysiol 2013; 24: 1021- 1027. 2Maltsev VA, Lakatta EG: The funny current in the context of the coupled-clock pacemaker cell system. Heart Rhythm 2013; 9: 302- 307. 3Yaniv Y, Sirenko S, Ziman BD, Spurgeon HA, Maltsev VA, Lakatta EG: New evidence for coupled clock regulation of the normal automaticity of sinoatrial nodal pacemaker cells: Bradycardic effects of ivabradine are linked to suppression of intracellular Ca cycling. J Mol Cell Cardiol 2013; 62: 80- 89. 4Bhuiyan ZA, van den Berg MP, van Tintelen JP, Bink-Boelkens MT, Wiesfeld AC, Alders M, Postma AV, van Langen I, Mannens MM, Wilde AA: Expanding spectrum of human RYR2-related disease: new electrocardiographic, structural, and genetic features. Circulation 2007; 116: 1569- 1576. 5Neco P, Torrente AG, Mesirca P, Zorio E, Liu N, Priori SG, Napolitano C, Richard S, Benitah JP, Mangoni ME, Gomez AM: Paradoxical effect of increased diastolic Ca(2+) release and decreased sinoatrial node activity in a mouse model of catecholaminergic polymorphic ventricular tachycardia. Circulation 2012; 126: 392- 401. 6Marger L, Mesirca P, Alig J, Torrente A, Dubel S, Engeland B, Kanani S, Fontanaud P, Striessnig J, Shin HS, Isbrandt D, Ehmke H, Nargeot J, Mangoni ME: Functional roles of Ca(v)1.3, Ca(v)3.1 and HCN channels in automaticity of mouse atrioventricular cells: insights into the atrioventricular pacemaker mechanism. Channels (Austin) 2011; 5: 251- 261. 7Baig SM, Koschak A, Lieb A, Gebhart M, Dafinger C, Nurnberg G, Ali A, Ahmad I, Sinnegger-Brauns MJ, Brandt N, Engel J, Mangoni ME, Farooq M, Khan HU, Nurnberg P, Striessnig J, Bolz HJ: Loss of Ca(v)1.3 (CACNA1D) function in a human channelopathy with bradycardia and congenital deafness. Nat Neurosci 2011; 14: 77- 84. Citing Literature Volume24, Issue12December 2013Pages E28-E29 ReferencesRelatedInformation" @default.
• W2146827984 created "2016-06-24" @default.
• W2146827984 creator A5049573849 @default.
• W2146827984 creator A5060742163 @default.
• W2146827984 date "2013-09-09" @default.
• W2146827984 modified "2023-09-27" @default.
• W2146827984 title "Pacemaker Gene Mutations, Bradycardia, Arrhythmias and the Coupled Clock Theory" @default.
• W2146827984 cites W2057934269 @default.
• W2146827984 cites W2060161469 @default.
• W2146827984 cites W2073256093 @default.
• W2146827984 cites W2093711619 @default.
• W2146827984 cites W2122638396 @default.
• W2146827984 cites W2143456942 @default.
• W2146827984 cites W2155488508 @default.
• W2146827984 doi "https://doi.org/10.1111/jce.12236" @default.
• W2146827984 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/3855211" @default.
• W2146827984 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/24015891" @default.
• W2146827984 hasPublicationYear "2013" @default.
• W2146827984 type Work @default.
• W2146827984 sameAs 2146827984 @default.
• W2146827984 citedByCount "7" @default.
• W2146827984 countsByYear W21468279842014 @default.
• W2146827984 countsByYear W21468279842015 @default.
• W2146827984 countsByYear W21468279842016 @default.
• W2146827984 countsByYear W21468279842019 @default.
• W2146827984 crossrefType "journal-article" @default.
• W2146827984 hasAuthorship W2146827984A5049573849 @default.
• W2146827984 hasAuthorship W2146827984A5060742163 @default.
• W2146827984 hasBestOaLocation W21468279842 @default.
• W2146827984 hasConcept C142724271 @default.
• W2146827984 hasConcept C161191863 @default.
• W2146827984 hasConcept C2777532764 @default.
• W2146827984 hasConcept C41008148 @default.
• W2146827984 hasConcept C71924100 @default.
• W2146827984 hasConcept C74909509 @default.
• W2146827984 hasConceptScore W2146827984C142724271 @default.
• W2146827984 hasConceptScore W2146827984C161191863 @default.
• W2146827984 hasConceptScore W2146827984C2777532764 @default.
• W2146827984 hasConceptScore W2146827984C41008148 @default.
• W2146827984 hasConceptScore W2146827984C71924100 @default.
• W2146827984 hasConceptScore W2146827984C74909509 @default.
• W2146827984 hasIssue "12" @default.
• W2146827984 hasLocation W21468279841 @default.
• W2146827984 hasLocation W21468279842 @default.
• W2146827984 hasLocation W21468279843 @default.
• W2146827984 hasLocation W21468279844 @default.
• W2146827984 hasOpenAccess W2146827984 @default.
• W2146827984 hasPrimaryLocation W21468279841 @default.
• W2146827984 hasRelatedWork W1994708679 @default.
• W2146827984 hasRelatedWork W2075980137 @default.
• W2146827984 hasRelatedWork W2093507182 @default.
• W2146827984 hasRelatedWork W2513888960 @default.
• W2146827984 hasRelatedWork W2794859701 @default.
• W2146827984 hasRelatedWork W2903735003 @default.
• W2146827984 hasRelatedWork W2912532158 @default.
• W2146827984 hasRelatedWork W2913088900 @default.
• W2146827984 hasRelatedWork W4283317414 @default.
• W2146827984 hasRelatedWork W871388907 @default.
• W2146827984 hasVolume "24" @default.
• W2146827984 isParatext "false" @default.
• W2146827984 isRetracted "false" @default.
• W2146827984 magId "2146827984" @default.
• W2146827984 workType "article" @default.