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- W2147315516 abstract "In healthy adults, (lymph)angiogenesis is limited to processes of repair (wound healing) and is restricted to the female reproductive tract (ovulatory cycle). In normal tissues, vascular quiescence is maintained by the dominant influence of endogenous angiogenesis inhibitors over angiogenic stimuli. In pathologic conditions, however, this delicate balance can be disrupted and lead to initiation and stimulation of blood or lymphatic vessel formation (‘angiogenic switch’). Neovascularization or angiogenesis -the formation of new blood vessels from pre-existing ones- is vital for the growth of tumors, providing a lifeline for sustenance and waste disposal. It is, therefore, tempting to speculate that interactions between osteotropic cancer cells and the (a)cellular bone marrow stoma lead to differential expression of (lymph)angiogenic factors and their inhibitors in the bone microenvironment, thus explaining -at least in part- the observed bone tropism by a limited number of carcinomas." @default.
- W2147315516 created "2016-06-24" @default.
- W2147315516 creator A5023166091 @default.
- W2147315516 creator A5064012002 @default.
- W2147315516 date "2004-01-01" @default.
- W2147315516 modified "2023-09-27" @default.
- W2147315516 title "Angiogenesis and Lymphangiogenesis in Metastatic Bone Disease: A Matter of Networking" @default.
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- W2147315516 doi "https://doi.org/10.1007/978-1-4020-2036-0_8" @default.
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