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• W2147696543 endingPage "4600" @default.
• W2147696543 startingPage "4593" @default.
• W2147696543 abstract "The TNFR family member BAFF-R facilitates peripheral B cell development, although it is unclear whether it promotes survival of B cells, or also initiates a differentiation program. We show that disruption of the BAFF-R encoding gene Tnfrsf13c in strain A/WySnJ mice causes a progressive decline in peripheral B cell numbers, beginning at the transitional 1 developmental stage and continuing through the mature peripheral B cell stage. Bcl-x(L) overexpression in A/WySnJ B cells decreased the turnover of transitional B cells, as determined by 5-bromo-2'-deoxyuridine labeling, and restored follicular B cell development. We conclude that the mutant A/WySnJ allele of Tnfrsf13c can be complemented through the survival signal provided by Bcl-x(L)." @default.
• W2147696543 created "2016-06-24" @default.
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• W2147696543 date "2003-05-01" @default.
• W2147696543 modified "2023-10-17" @default.
• W2147696543 title "Enforced<i>bcl-x</i><i>L</i>Gene Expression Restored Splenic B Lymphocyte Development in BAFF-R Mutant Mice" @default.
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• W2147696543 doi "https://doi.org/10.4049/jimmunol.170.9.4593" @default.
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