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- W2149267952 abstract "Most uremic subjects usually show muscle weakness and a decreased activity of daily living. Since the ability of the kidney to synthesize guanidinoacetic acid (GAA) decreases in uremia, the impaired metabolism of GAA may affect muscle energy metabolism, because creatine (CTN), an energy source of muscle, is a metabolite of GAA. CTN is produced from GAA in the liver, transfered to muscle tissue for energy storage, and stored as phospho-creatine (PC). In order to evaluate CTN metabolism in uremia, it is necessary to evaluate the muscle content of CTN and PC as well as that in blood. The usual method to measure CTN is an enzymatic spectrophotometric method and the sensitivity is not high enough to apply to muscular content. We investigated a more reliable method to measure CTN and PC in muscle tissue using HPLC.KeywordsMuscle TissueSodium Bicarbonate SolutionImpaired MetabolismQuadriceps FemorisMuscle ContentThese keywords were added by machine and not by the authors. This process is experimental and the keywords may be updated as the learning algorithm improves." @default.
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- W2149267952 date "1989-01-01" @default.
- W2149267952 modified "2023-09-27" @default.
- W2149267952 title "Measurement of Creatine and Phosphocreatine in Muscle Tissue Using HPLC" @default.
- W2149267952 doi "https://doi.org/10.1007/978-1-4613-0821-8_2" @default.
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