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• W2149663684 abstract "The title of this article has implications that differ greatly according to the reader's perspective: The clinician needs to know what the cell membrane has to do with elevated arterial pressure; the pharmacologist would like to know about functional abnormalities in this membrane; and the basic biologist is curious about whether these functional abnormalities can be accounted for by known differences at a molecular level. In an attempt to be global in our appeal, we have tried to address each of these three concerns. The elevated arterial pressure of hypertension is usually caused by an increased total peripheral resistance, and the magnitude of a pressor response to a standard pressor stimulus is uniformly greater than normal in hyperten­ sion. For both theoretical and applied reasons, it is important to know whether there are mechanistic differences responsible for these abnormalities among the various types of hypertension. At one level, all types of hypertension are the same in that in all of them arterial pressure and vascular responsiveness are elevated. Yet, as is evident in experimental hypertension, the initiating factors for genetic, renal, and mineralocorticoid hypertensions are entirely different. The relevant question is the relationship of sequences of events leading from the initiating factors to the final common outcomes. Where do these events converge? From the point of view of the current review it is important to ask whether all types of hypertension result from the same vascular changes. At one level this question can be given a convincing affirmative answer. In all types of hypertension, resistance vessels have walls that are thicker than normal. Folkow (1) has established that this characteristic not only increases vascular resistance by" @default.
• W2149663684 created "2016-06-24" @default.
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• W2149663684 date "1988-04-01" @default.
• W2149663684 modified "2023-10-12" @default.
• W2149663684 title "Vascular Smooth Muscle Membrane in Hypertension" @default.
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• W2149663684 doi "https://doi.org/10.1146/annurev.pa.28.040188.002133" @default.
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