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- W2150244264 abstract "Nitric oxide (NO) has been widely implicated in synaptic plasticity and memory formation. In studies of long-term potentiation (LTP), NO is thought to serve as a 'retrograde messenger' that contributes to presynaptic aspects of LTP expression. In this study, we examined the role of NO signaling in Pavlovian fear conditioning. We first show that neuronal nitric oxide synthase is localized in the lateral nucleus of the amygdala (LA), a critical site of plasticity in fear conditioning. We next show that NO signaling is required for LTP at thalamic inputs to the LA and for the long-term consolidation of auditory fear conditioning. Collectively, the findings suggest that NO signaling is an important component of memory formation of auditory fear conditioning, possibly as a retrograde signal that participates in presynaptic aspects of plasticity in the LA." @default.
- W2150244264 created "2016-06-24" @default.
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- W2150244264 creator A5047092516 @default.
- W2150244264 creator A5048552038 @default.
- W2150244264 creator A5050810881 @default.
- W2150244264 date "2005-07-01" @default.
- W2150244264 modified "2023-10-10" @default.
- W2150244264 title "Memory consolidation of Pavlovian fear conditioning requires nitric oxide signaling in the lateral amygdala" @default.
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- W2150244264 doi "https://doi.org/10.1111/j.1460-9568.2005.04209.x" @default.
- W2150244264 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/16029210" @default.
- W2150244264 hasPublicationYear "2005" @default.
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