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• W2151890693 abstract "Vibrio cholerae is the causative agent of the devastating diarrheal disease cholera. A number of regulatory pathways are involved in V. cholerae pathogenesis and antibiotic resistance. For example, there are over 40 LysR-family proteins in the V. cholerae genome, but most of their functions are unknown. In this study, we examine the role of VC2323 (TehA Vc ) and its divergently transcribed LysR-family regulator VC2324 (TehR Vc ) in V. cholerae pathogenesis. We found that in V. cholerae C6706, the expression of tehA Vc is dependent on TehR Vc . VC2323 (TehA Vc ), homologous to the Escherichia coli tellurite resistance protein (TehA Ec ), differs from TehA Ec in that TehA Vc has no noticeable role in tellurite resistance but instead contributes to chloramphenicol resistance. Interestingly, both tehA Vc and tehR Vc mutants were defective in colonization of infant mice. Though the expression of a key virulence gene tcpA was not affected in either of these mutants, tehA Vc mutants failed to attach to mouse intestinal surfaces in the presence of crude bile, suggesting a new role of the TehA Vc –TehR Vc pair in V. cholerae pathogenesis." @default.
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• W2151890693 date "2013-02-01" @default.
• W2151890693 modified "2023-10-15" @default.
• W2151890693 title "Role of a TehA homolog inVibrio choleraeC6706 antibiotic resistance and intestinal colonization" @default.
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• W2151890693 doi "https://doi.org/10.1139/cjm-2012-0673" @default.
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