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• W2151900613 endingPage "546" @default.
• W2151900613 startingPage "533" @default.
• W2151900613 abstract "Immune and inflammatory responses occurring in an injured nerve have been generally believed to contribute to the generation and maintenance of neuropathic pain. In this review, the authors demonstrate the upregulation of COX-2/prostaglandin E2, IL-6 and calcitonin gene-related peptide in invading macrophages and discuss possible mechanisms involved in their upregulation and how they contribute to the maintenance of neuropathic pain. By acting on nociceptors in dorsal root ganglion and local inflammatory cells via autocrine or paracrine pathways, these inflammatory mediators facilitate spontaneous ectopic activity and sustain nociceptive responses, an important mechanism underlying both ongoing and evoked neuropathic pain state. Targeting these mediators in injured nerve may provide novel therapeutic avenues to more successfully treat nerve injury-associated neuropathic pain." @default.
• W2151900613 created "2016-06-24" @default.
• W2151900613 creator A5055183128 @default.
• W2151900613 creator A5055901299 @default.
• W2151900613 date "2006-07-18" @default.
• W2151900613 modified "2023-10-12" @default.
• W2151900613 title "Targeting invading macrophage- derived PGE₂, IL-6 and calcitonin gene-related peptide in injured nerve to treat neuropathic pain" @default.
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