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- W2152384213 endingPage "1181" @default.
- W2152384213 startingPage "1165" @default.
- W2152384213 abstract "The glucocorticoid (GC) drugs are one of the most commonly prescribed and effective anti-inflammatory agents used for the treatment of many inflammatory disorders through their ability to attenuate phlogistic responses. The glucocorticoid receptor (GCR) primarily mediates GC actions via activation or repression of gene expression. GCs directly induce the expression of proteins displaying anti-inflammatory activities. However, the likely predominant effect of GCs is the repression of multiple inflammatory genes that invariably are overexpressed during nonresolving chronic inflammation. Although most GC actions are mediated through regulation of transcription, rapid nongenomic actions have also been reported. In addition, GCs modulate inflammatory cell survival, inducing apoptosis in immature thymocytes and eosinophils, while delaying constitutive neutrophil apoptosis. Importantly, GCs promote noninflammatory phagocytosis of apoptotic cell targets, a process important for the successful resolution of inflammation. Here, the effects and mechanisms of action of GC on inflammatory cell apoptosis and phagocytosis will be discussed." @default.
- W2152384213 created "2016-06-24" @default.
- W2152384213 creator A5004690424 @default.
- W2152384213 creator A5016151265 @default.
- W2152384213 creator A5050091992 @default.
- W2152384213 creator A5059920929 @default.
- W2152384213 date "2007-01-01" @default.
- W2152384213 modified "2023-09-23" @default.
- W2152384213 title "Effects of Glucocorticoids on Apoptosis and Clearance of Apoptotic Cells" @default.
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