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- W2153629315 abstract "Asthma is a chronic disease of the airways in which inflammation causes bronchial hyper-reactivity and consequent asthma attacks triggered by various stimuli. The bronchospasm attacks are usually relieved by short-acting β2 agonists, and inflammation and bronchial hyper-reactivity are reduced by maintenance therapy and, in particular, by inhaled corticosteroids. In milder asthma subjects, airway inflammation is dominated by eosinophils, whereas in more severe asthma increased neutrophil counts were detected. In severe/refractory asthma, TNF-α is known to play a role in the maintenance of neutrophilic inflammation and of bronchial hyper-responsiveness and is not influenced by corticosteroid therapy. Etanercept, a TNF-α-blocking agent, could represent one of the potential therapies for refractory asthma based on demonstrated safety and efficacy." @default.
- W2153629315 created "2016-06-24" @default.
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- W2153629315 date "2006-09-21" @default.
- W2153629315 modified "2023-09-23" @default.
- W2153629315 title "Etanercept for refractory asthma therapy" @default.
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- W2153629315 doi "https://doi.org/10.1517/13543784.15.10.1279" @default.
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