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- W2153957488 endingPage "58" @default.
- W2153957488 startingPage "743" @default.
- W2153957488 abstract "To evaluate the impact of an antibiotic, minocycline, on several immune parameters in response to radiation in a mouse model.C57BL/6 mice were treated with minocycline (i.p.) for 5 days, beginning immediately before radiation with 1-3 Gy (60)Co γ-rays. Spleen and blood were collected on day 4 post-irradiation. Cell populations were determined in the blood and spleen. Splenocytes were activated with anti-CD3 antibody for 48 h and cytokines were quantified.Minocycline increased the counts and/or percentages of splenic macrophages, granulocytes, natural killer, T- and CD8(+) T-cells (p<0.05 versus radiation alone). Minocycline significantly increased the expression of interleukin-1α and β, which are radioprotective, as well as the ones of granulocyte-macrophage colony-stimulating factor and granulocyte colony-stimulating factor, which accelerate neutrophil recovery (p<0.05 versus radiation alone), while suppressing cytokines that could prevent hematopoiesis, e.g. macrophage inflammatory protein-1α, tumor necrosis factor-α and interferon-γ.These data indicate that minocycline should be further tested for use in restoration of the hematopoietic system after radiation exposure." @default.
- W2153957488 created "2016-06-24" @default.
- W2153957488 creator A5009118049 @default.
- W2153957488 creator A5028857100 @default.
- W2153957488 creator A5060817429 @default.
- W2153957488 date "2012-09-06" @default.
- W2153957488 modified "2023-09-23" @default.
- W2153957488 title "Analysis of minocycline as a countermeasure against acute radiation syndrome." @default.
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- W2153957488 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/22949587" @default.