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• W2154497039 abstract "Background: Environmental exposures, ranging from perchlorate in rocket fuel to polychlorinated biphenols, have been shown to influence thyroid function. Although most of these agents are associated with reduced thyroid hormone levels or impaired thyroid hormone action, a number of environmental exposures confer an increased risk of autoimmune thyroid disease. Summary: Factors that increase autoimmune thyroid disease risk include radiation exposure, both from nuclear fallout and medical radiation, increased iodine intake, as well as several contaminants in the environment that influence the thyroid. Although ∼70% of the risk for developing autoimmune thyroid disease is attributable to genetic background, environmental triggers are thought to play a role in the development of autoimmune thyroid disease in susceptible individuals. Conclusions: Understanding the association of environmental agents with thyroid dysfunction can be utilized to reduce the risk to populations. Knowledge of the specific factors that trigger autoimmune thyroid disease and their mode of action, however, may also inform risk reduction in the individual patient. These factors are especially relevant for those at increased risk of autoimmune thyroid disease based on family history." @default.
• W2154497039 created "2016-06-24" @default.
• W2154497039 creator A5049222616 @default.
• W2154497039 date "2010-07-01" @default.
• W2154497039 modified "2023-09-30" @default.
• W2154497039 title "Environmental Exposures and Autoimmune Thyroid Disease" @default.
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• W2154497039 doi "https://doi.org/10.1089/thy.2010.1636" @default.
• W2154497039 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/2935336" @default.
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