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• W2154796202 endingPage "1880" @default.
• W2154796202 startingPage "1868" @default.
• W2154796202 abstract "Experiments presented in this report demonstrate that specificity of the Ly1+ T cell proliferative response to NP-modified Ig is controlled by Igh-C-linked genes. In addition, we describe the mechanism whereby Igh-C-encoded molecules influence Ly1+ T cell activity. We show that Igh-C-linked control of T cell responses to NP-modified Ig is a secondary consequence of naturally acquired tolerance for self Ig. Unresponsiveness to self Ig is not due to a defect expressed functionally at the level of the antigen-presenting cell, nor is it associated with active suppression. These results suggest that tolerance for self Ig at the level of the Ly1+ T cell is due to functional deletion of Ly1+ T cell clones specific for self Ig. The possibility is considered that regulatory effects mediated by passively administered antibodies may in part be due to induction of Ly1+ T cell tolerance for self Ig." @default.
• W2154796202 created "2016-06-24" @default.
• W2154796202 creator A5067921282 @default.
• W2154796202 date "1983-12-01" @default.
• W2154796202 modified "2023-09-27" @default.
• W2154796202 title "Tolerance for self IG at the level of the Ly1+ T cell." @default.
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• W2154796202 doi "https://doi.org/10.1084/jem.158.6.1868" @default.
• W2154796202 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/2187155" @default.
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• W2154796202 hasPublicationYear "1983" @default.
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