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- W2154803724 endingPage "490" @default.
- W2154803724 startingPage "475" @default.
- W2154803724 abstract "Acquired myasthenia gravis (MG) is an autoimmune disorder of the neuromuscular junction in which patients experience fluctuating skeletal muscle weakness that often affects selected muscle groups preferentially. The target of the autoimmune attack in most cases is the skeletal muscle acetylcholine receptor (AChR), but in others, non-AChR components of the neuromuscular junction, such as the muscle-specific receptor tyrosine kinase, are targeted. The pathophysiological result is muscle endplate dysfunction and consequent fatigable muscle weakness. Clinical presentations vary substantially, both for anti-AChR positive and negative MG, and accurate diagnosis and selection of effective treatment depends on recognition of less typical as well as classic disease phenotypes. Accumulating evidence suggests that clinical MG subgroups might respond differently to treatment. In this Review, we provide current information about the epidemiology, immunopathogenesis, clinical presentations, diagnosis, and treatment of MG, including emerging therapeutic strategies." @default.
- W2154803724 created "2016-06-24" @default.
- W2154803724 creator A5057735384 @default.
- W2154803724 creator A5076681315 @default.
- W2154803724 date "2009-05-01" @default.
- W2154803724 modified "2023-09-30" @default.
- W2154803724 title "Autoimmune myasthenia gravis: emerging clinical and biological heterogeneity" @default.
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