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• W2155240779 startingPage "29" @default.
• W2155240779 abstract "Eosinophilic esophagitis (EoE) is a newly recognized disease and is an emerging entity throughout developing and developed countries, including the United States. Therefore, understanding the causes, natural history, diagnosis, and management is important for future therapeutic interventions. The pathogenesis of EoE is still not clear, but a growing body of evidence has established that this condition represents a T-cell-mediated immune response involving several proinflammatory mediators and chemoattractants known to regulate eosinophilic accumulation in the esophagus, such as IL-4, IL-5, IL-3 and eotaxin-1, -2, and -3. Determining the mechanism or mechanisms through which human esophageal-derived factors ultimately induce the functional abnormalities observed, and to which antigens patients who have EoE are sensitized that lead to the manifestation of symptoms, is of significant interest." @default.
• W2155240779 created "2016-06-24" @default.
• W2155240779 creator A5081548576 @default.
• W2155240779 date "2009-02-01" @default.
• W2155240779 modified "2023-09-30" @default.
• W2155240779 title "Mechanism of Eosinophilic Esophagitis" @default.
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• W2155240779 doi "https://doi.org/10.1016/j.iac.2008.09.010" @default.
• W2155240779 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/2650237" @default.
• W2155240779 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/19141339" @default.
• W2155240779 hasPublicationYear "2009" @default.
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