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• W2156073971 abstract "Experimental studies have demonstrated the involvement of angiogenesis-related factors in the progression of chronic kidney disease (CKD). There have so far been no reports investigating the distribution and clinical roles of Vasohibin-1 (VASH-1), a negative feedback regulator of angiogenesis, in CKD. We recruited 54 Japanese CKD patients and 6 patients who had normal renal tissues excised due to localized renal cell carcinoma. We evaluated the correlations between the renal expression level of VASH-1 and the clinical/histological parameters. VASH-1 was observed in renal endothelial/mesangial cells, crescentic lesions and interstitial inflammatory cells. Significant positive correlations were observed between 1) crescent formation and the number of VASH-1+ cells in the glomerulus (r＝0.48, p＝0.001) or cortex (r＝0.64, p＜0.0001), 2) interstitial cell infiltration and the number of VASH-1+ cells in the cortex (r＝0.34, p＝0.02), 3) the glomerular VEGFR-2+ area and the number of VASH-1+ cells in the glomerulus (r＝0.44, p＝0.01) or medulla (r＝0.63, p＝0.01). These results suggest that the renal levels of VASH-1 may be affected by local inflammation, crescentic lesions and VEGFR-2." @default.
• W2156073971 created "2016-06-24" @default.
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• W2156073971 date "2014-01-01" @default.
• W2156073971 modified "2023-09-23" @default.
• W2156073971 title "Renal distribution of Vasohibin-1 in patients with chronic kidney disease." @default.
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• W2156073971 doi "https://doi.org/10.18926/amo/52788" @default.
• W2156073971 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/25145408" @default.
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