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• W2157029758 abstract "Monocytes from patients with sepsis have a reduced capacity to produce cytokines, a state referred to as immunoparalysis. To determine whether polymorphonuclear leukocytes (PMNL) can be rendered hyporesponsive, PMNL from 6 healthy volunteers intravenously challenged with lipopolysaccharide (LPS; 4 ng/kg) were stimulated ex vivo with heat-killed bacteria or LPS, and the release of the CXC chemokines interleukin-8, epithelial-derived neutrophil attractant—78, and growth-related oncogen—α was measured. At 1 and 2 h after LPS administration in vivo, PMNL produced fewer CXC chemokines after stimulation with bacteria or LPS (all P <.05). Serum obtained 2 h after in vivo administration of LPS did not influence chemokine production by PMNL from 6 healthy volunteers not previously exposed to LPS. Thus, intravenous injection of LPS induces a refractory state of PMNL that is not caused by soluble factors produced in response to in vivo exposure to LPS." @default.
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• W2157029758 date "2000-10-01" @default.
• W2157029758 modified "2023-10-18" @default.
• W2157029758 title "Reduced Ex Vivo Chemokine Production by Polymorphonuclear Cells after In Vivo Exposure of Normal Humans to Endotoxin" @default.
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• W2157029758 doi "https://doi.org/10.1086/315840" @default.
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