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- W2157041107 abstract "Early-life programming is becoming an established concept that states that the environment during early development affects health and disease in adulthood, probably via epigenetic mechanisms such as DNA methylation, histone modifications, RNA silencing, or a combination. Accumulating evidence suggests that nutrition during pregnancy and early postnatal life is one of the most important environmental cues that programs microbiological, metabolic, and immunologic development. The neonatal period is crucial for the early microbial colonization of the almost sterile gastrointestinal tract of the newborn infant. These first colonizers play an important role in host health because they are involved in nutritional, immunologic, and physiologic functions. Evidence from animal and human studies indicates that the composition of the gut microbiota has an effect on body composition, digestion, and metabolic homeostasis. Furthermore, the functionality of the metabolism develops after birth when the newborn is first exposed to nutrition via the gastrointestinal tract. Exposure to environmental microbial components is also suggested to have a key role in the maturation process of the immune system, and in turn the immune system shapes the composition of the microbiota. Therefore, the use of nutritional strategies to program the microbiota composition to favor a more beneficial bacterial population and to support the development of the metabolic and immune systems may provide a good opportunity to prevent later health problems such as obesity, diabetes, and allergy." @default.
- W2157041107 created "2016-06-24" @default.
- W2157041107 creator A5009298996 @default.
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- W2157041107 creator A5045824083 @default.
- W2157041107 creator A5067535218 @default.
- W2157041107 creator A5080103519 @default.
- W2157041107 date "2013-08-01" @default.
- W2157041107 modified "2023-10-12" @default.
- W2157041107 title "Relevance of pre- and postnatal nutrition to development and interplay between the microbiota and metabolic and immune systems,," @default.
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