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• W2157261330 abstract "<h3>Background</h3> The spatial and temporal pattern of excessive disease occurrence, termed<i>clustering</i>, may provide clues about the underlying etiology. <h3>Objective</h3> To report the occurrence of 3 clusters of Parkinson disease (PD) in Canada. <h3>Design and Patients</h3> We determined the population groups containing the clusters, geographical limits, and duration of exposure to the specific environments. We tested whether there was an excessive presence of Parkinson disease by calculating the probability of the observed cases occurring under the null hypothesis that the disease developed independently and at random in cluster subjects. Results of genetic testing for mutations in the α-synuclein, parkin, tau genes, and spinocerebellar ataxia genes (<i>SCA2</i>and<i>SCA3</i>) were negative. <h3>Results</h3> The probabilities of random occurrence (<i>P</i>values) in the 3 clusters were<i>P</i>= 7.9 × 10⁻⁷for cluster 1,<i>P</i>= 2.6 × 10⁻⁷for cluster 2, and<i>P</i>= 1.5 × 10⁻⁷for cluster 3. <h3>Conclusions</h3> Our findings indicate an important role for environmental causation in Parkinson disease. A possible role exists for environmental factors such as viral infection and toxins in the light of current evidence." @default.
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• W2157261330 date "2004-07-01" @default.
• W2157261330 modified "2023-09-27" @default.
• W2157261330 title "Clustering of Parkinson Disease" @default.
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• W2157261330 doi "https://doi.org/10.1001/archneur.61.7.1057" @default.
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