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- W2158223248 endingPage "481" @default.
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- W2158223248 abstract "In mammalian cells, there is an extensive and continuous exchange of mitochondrial DNA (mtDNA) and its products between mitochondria. This mitochondrial complementation prevents individuals from expression of respiration deficiency caused by mutant mtDNAs. Thus, the presence of mitochondrial complementation does not support the generally accepted mitochondrial theory of aging, which proposes that accumulation of somatic mutations in mtDNA is responsible for age-associated mitochondrial dysfunction. Moreover, the presence of mitochondrial complementation enables gene therapy for mitochondrial diseases using nuclear transplantation of zygotes." @default.
- W2158223248 created "2016-06-24" @default.
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- W2158223248 creator A5033614902 @default.
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- W2158223248 date "2006-05-01" @default.
- W2158223248 modified "2023-10-15" @default.
- W2158223248 title "Mitochondrial dynamics and aging: Mitochondrial interaction preventing individuals from expression of respiratory deficiency caused by mutant mtDNA" @default.
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- W2158223248 doi "https://doi.org/10.1016/j.bbamcr.2006.03.001" @default.
- W2158223248 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/16624428" @default.
- W2158223248 hasPublicationYear "2006" @default.
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