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• W2160839536 abstract "The cells of innate and adaptive immunity, although activated by different ligands, engage in cross talk to ensure a successful immune outcome. Toll-like receptors (TLRs) are key components of the innate immune system and have the ability to detect microbial infection and trigger host defence responses. Otherwise, human T lymphocytes are able to produce most TLRs. Thus, we analyze the capability of some TLR ligands to modulate the function of highly-purified CD4+ T cells. We found that agents acting via TLRs (poly I:C, a TLR3 ligand; flagellin, a TLR5 ligand; and R848, a TLR7/8 ligand) are able to regulate the expression of costimulatory molecules both on purified antigen presenting cells and on purified T lymphocytes. Moreover, the activation mediated by TLRs determines a kinetic expression of B7-family members such as through an inhibition of T lymphocytes delayed proliferation. These findings suggest a functional role of some invading microorganisms in regulating acquired immunity." @default.
• W2160839536 created "2016-06-24" @default.
• W2160839536 creator A5018655605 @default.
• W2160839536 creator A5024875387 @default.
• W2160839536 creator A5088351858 @default.
• W2160839536 date "2009-02-10" @default.
• W2160839536 modified "2023-09-27" @default.
• W2160839536 title "Stimulation of Human CD4+ T Lymphocytes via TLR3, TLR5 and TLR7/8 Up-Regulates Expression of Costimulatory and Modulates Proliferation" @default.
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• W2160839536 doi "https://doi.org/10.2174/1874285800903010001" @default.
• W2160839536 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/2656776" @default.
• W2160839536 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/19294011" @default.
• W2160839536 hasPublicationYear "2009" @default.
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