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- W2160892014 abstract "Chronic lymphocytic leukemia (CLL) cells display features consistent with a defect in apoptosis and exhibit prolonged survival in vivo. Survival of these malignant cells is influenced by interactions with non-leukemic cells located in permissive niches in lymphoid organs. Leukemic cells subvert the normal architecture of the lymphoid organs, recruiting stromal cells, dendritic cells and T lymphocytes, all reported as playing active roles in the survival and proliferation of CLL. The same survival-promoting environment also rescues/protects leukemic cells from cytotoxic therapies, giving way to disease relapse. This review summarizes and discusses current knowledge about the intricate network of soluble and cell-bound signals regulating the life and death of CLL cells in different districts. At the same time, it seeks to hone in on which discrete molecular elements are best suited as targets for treating this still incurable disease." @default.
- W2160892014 created "2016-06-24" @default.
- W2160892014 creator A5007447330 @default.
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- W2160892014 creator A5065756392 @default.
- W2160892014 creator A5068535005 @default.
- W2160892014 date "2013-01-01" @default.
- W2160892014 modified "2023-10-18" @default.
- W2160892014 title "Targeting the microenvironment in chronic lymphocytic leukemia offers novel therapeutic options" @default.
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