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• W2163496345 abstract "Infections with Salmonella enterica serovar Typhimurium and Shigella flexneri result in mucosal inflammation in response to epithelial cell invasion and macrophage cytotoxicity. These processes are mediated by type III secretion systems encoded in homologous virulence loci in the two species, namely, Salmonella pathogenicity island 1 (SPI-1), carried in the genome, and the Shigella entry region (SER), carried in a large virulence plasmid. Here we show that SPI-1 can functionally complement a deletion of SER in S. flexneri, restoring invasion of epithelial cells, macrophage cytotoxicity, and phagosomal escape. Furthermore, S. flexneri phagosomal escape requires the SER and another gene(s) carried on the large virulence plasmid. We demonstrate that the processes of invasion and phagosomal escape can be uncoupled in S. flexneri." @default.
• W2163496345 created "2016-06-24" @default.
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• W2163496345 date "2007-10-01" @default.
• W2163496345 modified "2023-10-03" @default.
• W2163496345 title "<i>Shigella flexneri</i> Phagosomal Escape Is Independent of Invasion" @default.
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• W2163496345 doi "https://doi.org/10.1128/iai.00454-07" @default.
• W2163496345 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/2044536" @default.
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