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- W2164163902 abstract "Excessive energy storage of adipose tissue makes contribution to the occurrence and progression of obesity, which accompanies with multiple adverse complications, such as metabolic syndrome, cardiovascular diseases. It is well known that apolipoprotein E, as a component of lipoproteins, performs a key role in maintaining plasma lipoproteins homeostasis. Interestingly, apolipoprotein E is highly expressed in adipocyte and has positive relation with body fat mass. Apolipoprotein E knock-out mice show small fat mass compared to wild type mice. Moreover, adipocyte deficiency in apolipoprotein E shows impaired lipoproeteins internalization and triglyceride accumulation. Apolipopreotein E-deficient lipoproteins can not induce preadipocyte to form round full-lipid adipocyte, whereas apolipoprotein E-containing lipoproteins can. This article mainly reviews the modulation of apolipoprotein E synthesized by adipocyte and apolipoprotein E carried on lipoproteins in adipocyte triglyceride content." @default.
- W2164163902 created "2016-06-24" @default.
- W2164163902 creator A5017360203 @default.
- W2164163902 creator A5064435064 @default.
- W2164163902 date "2014-08-23" @default.
- W2164163902 modified "2023-10-10" @default.
- W2164163902 title "Apolipoprotein E synthesized by adipocyte and apolipoprotein E carried on lipoproteins modulate adipocyte triglyceride content" @default.
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- W2164163902 doi "https://doi.org/10.1186/1476-511x-13-136" @default.
- W2164163902 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/4156606" @default.
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