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- W2165884537 abstract "Aims: Clinical and pre-clinical studies have demonstrated that alcohol abuse, aging, diabetes and rheumatoid arthritis are associated with increased risk of fractures compounded with impaired fracture repair. We note that these and other pathologies are characterized by chronic inflammation (CI) as a risk factor. How these CI pathologies inhibit bone repair is unclear, but one candidate mediator is endotoxin (lipopolysaccharide/LPS). LPS promotes inflammation and is present in increased serum concentrations in some inflammatory conditions. The distraction osteogenesis (DO) model developed in this laboratory provides the opportunity to isolate and study the effects of CI on direct bone formation during bone regeneration. The aim of this study was to determine whether endotoxin at concentrations that mimic levels reported" @default.
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- W2165884537 date "2012-01-10" @default.
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- W2165884537 title "Metabolic Endotoxemia and Inhibition of Direct Bone Regeneration: A Pilot Study" @default.
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- W2165884537 doi "https://doi.org/10.9734/bjmmr/2012/1547" @default.
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