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- W2166601264 abstract "Background Close to half of the human genome is derived from transposable elements (TEs), and some TE families continue to generate new insertions through RNA-mediated mechanisms. Due to its mutagenic potential, such retrotransposition is normally suppressed by epigenetic and post-transcriptional mechanisms. However, the epigenetic and regulatory disruptions commonly observed in cancers may allow for TE activation, and a few examples have been reported in lung and colon cancer previously." @default.
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- W2166601264 date "2012-10-01" @default.
- W2166601264 modified "2023-09-27" @default.
- W2166601264 title "Analysis of somatic retrotransposition in human cancers" @default.
- W2166601264 doi "https://doi.org/10.1186/1753-6561-6-s6-o23" @default.
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