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• W2167081558 abstract "Cannabinoid receptors of type 1 and 2 (CB₁ and CB₂), endogenous ligands that activate them (endocannabinoids), and mechanisms for endocannabinoid biosynthesis and inactivation have been identified in the gastrointestinal system. Activation of CB₁receptors by endocannabinoids produces relaxation of the lower oesophageal sphincter and inhibition of gastric acid secretion, intestinal motility, and fluid stimulated secretion. However, stimulation of cannabinoid receptors impacts on gastrointestinal functions in several other ways. Recent data indicate that the endocannabinoid system in the small intestine and colon becomes over stimulated during inflammation in both animal models and human inflammatory disorders. The pathological significance of this “endocannabinoid overactivity” and its possible exploitation for therapeutic purposes are discussed here." @default.
• W2167081558 created "2016-06-24" @default.
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• W2167081558 date "2006-10-01" @default.
• W2167081558 modified "2023-09-25" @default.
• W2167081558 title "Endocannabinoid overactivity and intestinal inflammation" @default.
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• W2167081558 doi "https://doi.org/10.1136/gut.2005.090472" @default.
• W2167081558 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/1856409" @default.
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