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• W2167123485 abstract "Abstract Background The prevalence, morbidity, and mortality of inflammatory lung diseases such as asthma, chronic obstructive pulmonary disease (COPD) and cystic fibrosis (CF) are increasing in women. There is a dearth of data on the biological mechanisms to explain such observations. However, some large epidemiologic studies suggest that lung function fluctuates during the menstrual cycle in female patients with airways disease but not in women without disease, suggesting that circulating estradiol and progesterone may be involved in this process. Discussion In asthma, estradiol shuttles adaptive immunity towards the T H 2 phenotype while in smokers estrogens may be involved in the generation of toxic intermediate metabolites in the airways of female smokers, which may be relevant in COPD pathogenesis. In CF, estradiol has been demonstrated to up-regulate MUC5B gene in human airway epithelial cells and inhibit chloride secretion in the airways. Progesterone may augment airway inflammation. Summary Taken together, clinical and in-vivo data have demonstrated a sex-related difference in that females may be more susceptible to the pathogenesis of lung diseases. In this paper, we review the effect of female sex hormones in the context of these inflammatory airway diseases." @default.
• W2167123485 created "2016-06-24" @default.
• W2167123485 creator A5019087640 @default.
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• W2167123485 date "2011-06-03" @default.
• W2167123485 modified "2023-10-04" @default.
• W2167123485 title "The role of female hormones on lung function in chronic lung diseases" @default.
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• W2167123485 doi "https://doi.org/10.1186/1472-6874-11-24" @default.
• W2167123485 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/3129308" @default.
• W2167123485 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/21639909" @default.
• W2167123485 hasPublicationYear "2011" @default.
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