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- W2167233442 abstract "ABSTRACT The block toward human immunodeficiency virus type 1 (HIV-1) infection of dendritic cells (DCs) can be relieved by Vpx (viral protein X), which degrades sterile alpha motif-hydroxylase domain 1 (SAMHD1) or by exogenously added deoxynucleosides (dNs), lending support to the hypothesis that SAMHD1 acts by limiting deoxynucleoside triphosphates (dNTPs). This notion has, however, been questioned. We show that while dNs and Vpx increase the infectivity of HIV-1, only the latter restores the infectivity of a simian immunodeficiency virus of macaques variant, SIV MAC ΔVpx virus. This distinct behavior seems to map to CA, suggesting that species-specific CA interactors modulate infection of DCs." @default.
- W2167233442 created "2016-06-24" @default.
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- W2167233442 date "2015-04-01" @default.
- W2167233442 modified "2023-10-03" @default.
- W2167233442 title "The Susceptibility of Primate Lentiviruses to Nucleosides and Vpx during Infection of Dendritic Cells Is Regulated by CA" @default.
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- W2167233442 doi "https://doi.org/10.1128/jvi.03315-14" @default.
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- W2167233442 hasPublicationYear "2015" @default.
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