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• W2167472716 abstract "Chronic inflammation of the gastric epithelium has been associated with the pathogenesis of gastric cancer, as it was postulated by Corea's model of gastric carcinogenesis. Helicobacter pylori (Hp) regulates this inflammatory process and promotes gastric carcinogenesis through induction of gene mutations and protein modulation. Recent data raise the cancer stem cell hypothesis, which implies a central role of multipotent cancer cells in oncogenesis of various solid tumors. This review provides a synopsis of gastric cancer initiation and promotion through Hp and stem cell signaling pathways. The expanding research field of Hp-related cancer stem cell biology may offer novel implications for future treatment of upper gastrointestinal cancer." @default.
• W2167472716 created "2016-06-24" @default.
• W2167472716 creator A5043566872 @default.
• W2167472716 creator A5052332696 @default.
• W2167472716 creator A5054200484 @default.
• W2167472716 creator A5072218541 @default.
• W2167472716 date "2011-04-01" @default.
• W2167472716 modified "2023-09-25" @default.
• W2167472716 title "Upper Gastrointestinal Carcinogenesis: H. pylori and Stem Cell Cross-Talk" @default.
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• W2167472716 doi "https://doi.org/10.1016/j.jss.2010.02.012" @default.
• W2167472716 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/20452613" @default.
• W2167472716 hasPublicationYear "2011" @default.
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