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- W2170394948 abstract "We read with interest the article by Wiest et al about spontaneous bacterial peritonitis guidelines.1 The authors point out the relevance of pathological bacterial translocation (BT) from the gut to mesenteric lymph nodes (MLN) as the pathophysiological hallmark of spontaneous bacterial peritonitis in cirrhosis, and the need of further research in the factors involved in BT. The study of BT, ascites and related immune system abnormalities in carbon tetrachloride (CCl4) cirrhosis is usually conducted in rats. This model has proved useful for research into the pathogenesis and role of the systemic pro-inflammatory response associated with cirrhosis and its link to BT.2–4 We recently showed that modulation of the immune system response in cirrhotic rats …" @default.
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- W2170394948 date "2012-07-19" @default.
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- W2170394948 title "Systemic inflammation in absence of gut bacterial translocation in C57BL/6 mice with cirrhosis" @default.
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- W2170394948 doi "https://doi.org/10.1136/gutjnl-2012-302920" @default.
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