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• W2170484893 abstract "β2-adrenergic agonists exhibit antipermeability effects in the airways. However, it is not known whether β2-agonists have this beneficial effect in airway mucosa that is already inflamed. We evaluated the effects of two inhaled β2-agonists, salbutamol and formoterol, on the histamine-induced bronchoconstriction and plasma extravasation in the airways of guinea pigs with or without ozone exposure. Total pulmonary resistance (RL) was measured before and after histamine inhalation in anesthetized animals that were pretreated with inhaled salbutamol, formoterol, or saline. Plasma extravasation in the airways was measured using Evans blue dye. In the control animals not exposed to ozone, salbutamol and formoterol each significantly reduced both the histamine-induced bronchoconstriction and the plasma extravasation in the trachea and main bronchi. In the ozone-exposed animals, the increase in RL after histamine was greater than that in control animals. Salbutamol and formoterol each significantly reduced histamine-induced bronchoconstriction, even in the ozone-exposed animals. Salbutamol did not affect the histamine-induced plasma extravasation, whereas formoterol reduced the plasma extravasation in the main bronchi, but not in the trachea, of the animals exposed to ozone. These results suggest that the anti-inflammatory properties of formoterol in inflamed airways may contribute to the beneficial effects in the treatment of airway inflammation." @default.
• W2170484893 created "2016-06-24" @default.
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• W2170484893 date "1997-09-01" @default.
• W2170484893 modified "2023-10-18" @default.
• W2170484893 title "Effect of β₂-agonists on Histamine-induced Airway Microvascular Leakage in Ozone-exposed Guinea Pigs" @default.
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• W2170484893 doi "https://doi.org/10.1164/ajrccm.156.3.9606019" @default.
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