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- W2170822974 startingPage "287" @default.
- W2170822974 abstract "Despite novel surgical therapies for the treatment of atherosclerosis, restenosis continues to be a significant impediment to the long-term success of vascular interventions. Transforming growth factor-beta (TGF-β), a family of cytokines found to be up-regulated at sites of arterial injury, has long been implicated in restenosis; a role that has largely been attributed to TGF-β-mediated vascular fibrosis. However, emerging data indicate that the role of TGF-β in intimal thickening and arterial remodeling, the critical components of restenosis, is complex and multidirectional. Recent advancements have clarified the basic signaling pathway of TGF-β, making evident the need to redefine the precise role of this family of cytokines and its primary signaling pathway, Smad, in restenosis. Unraveling TGF-β signaling in intimal thickening and arterial remodeling will pave the way for a clearer understanding of restenosis and the development of innovative pharmacological therapies." @default.
- W2170822974 created "2016-06-24" @default.
- W2170822974 creator A5002465662 @default.
- W2170822974 creator A5012140656 @default.
- W2170822974 creator A5090815103 @default.
- W2170822974 date "2011-05-01" @default.
- W2170822974 modified "2023-09-27" @default.
- W2170822974 title "TGF-β and Restenosis Revisited: A Smad Link" @default.
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