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- W2174305377 abstract "Significance Glucocorticoids are primary stress and immune mediators of several fundamental processes, including reproduction and development. However, the direct actions of glucocorticoid signaling in the uterus are largely unknown. We have discovered that the glucocorticoid receptor (GR) in the mouse uterus is essential for normal fertility. Female mice lacking GR specifically in the uterus are subfertile due to defects in implantation related to inadequate remodeling of the endometrial stroma. Moreover, the deficiency in uterine glucocorticoid signaling leads to aberrant gene regulation during tissue remodeling events. Our findings demonstrate previously unrecognized functions of GR in both uterine physiology and fertility." @default.
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- W2174305377 date "2015-11-23" @default.
- W2174305377 modified "2023-09-25" @default.
- W2174305377 title "Uterine glucocorticoid receptors are critical for fertility in mice through control of embryo implantation and decidualization" @default.
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- W2174305377 doi "https://doi.org/10.1073/pnas.1508056112" @default.
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