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- W2174668721 abstract "Intense and/or prolonged stress can trigger a large class of psychiatric conditions which includes schizophrenic psychoses, anxiety spectrum disorders and depression. Data of the last decade are beginning to recognize inflammation as a factor shared by this apparently inhomogeneous set of illnesses. An abnormally large percentage of patients suffering from these conditions also share nicotine-(ab)use as common habit, suggestive of an attempt at self-medication [1]. Evidence of the last two decades shows that activation of the anti-inflammatory reflex, mediated by activation of nicotinic receptors on immune cells, decreases the release of proinflammatory cytokines [2]. The pharmacological or electrophysiological activation of nicotinic receptors has unexplored potential for the treatment of such conditions, particularly for patients refractory to traditional illness-specific treatment. In this editorial, we will discuss the hypothesis that, since an inflammatory component underlies synaptic impairment associated with psychiatric disease, the use of nicotine agonists potentially ameliorates illness – from psychosis to depression through anxiety and bipolar disease – by decreasing inflammation." @default.
- W2174668721 created "2016-06-24" @default.
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- W2174668721 date "2015-07-01" @default.
- W2174668721 modified "2023-09-27" @default.
- W2174668721 title "Nicotine for psychiatric disease: from nuisance to novel treatment?" @default.
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- W2174668721 doi "https://doi.org/10.4155/fmc.15.67" @default.
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