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• W2174714364 abstract "Systemic lupus erythematosus (SLE) has a multifactorial pathogenesis that includes genetic and environmental precipitating factors (reviewed in ref. 1). Considerable attention has been focused on the identity of genes that enhance the susceptibility or resistance to SLE, the identity of antigenic determinants that trigger the disease, the molecular mechanisms of T-cell activation that result in survival or apoptosis, cytokines that determine T-cell function, and the properties of the autoantibody-forming B cells. Although it is well recognized that the primary role of certain lymphocytes is to downregulate immune responses, progress in elucidating their precise identity and the mechanisms involved in generating these cells has been slow. Both major histocompatibility complex (MHC)-restricted, antigen-specific and MHC nonrestricted, antigen nonspecific T cells that downregulate antibody production have been described. The identity and characterization of antigen nonspecific regulatory cells has been a principal interest of our group. Whereas Stohl has focused his attention on the role of cytolytic T cells in SLE (see Chapter 18), we have contemplated how antigen nonspecific suppressor cells are generated in healthy subjects." @default.
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• W2174714364 date "1999-01-01" @default.
• W2174714364 modified "2023-09-27" @default.
• W2174714364 title "Natural Killer Cells and CD8+ T Cells in the Downregulation of Antibody Production in Healthy Subjects and SLE" @default.
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• W2174714364 doi "https://doi.org/10.1007/978-1-59259-703-1_19" @default.
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