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• W2177821000 startingPage "R8" @default.
• W2177821000 abstract "The hallmark neuroendocrine response to stress is increased plasma ACTH. Inhibition of neurons in the region of the dorsomedial hypothalamus (DMH) attenuates experimental air stress-induced elevation of heart rate (HR), mean arterial pressure (MAP), and plasma ACTH. We hypothesized that, under basal conditions, stimulation of the DMH would mimic the neuroendocrine and cardiovascular response to air stress. We examined the effects of unilateral microinjection (100-nl vol) of bicuculline methiodide (BMI, 10 pmol), kainate (KA, 1 or 3 pmol), and N-methyl-D-aspartate (5 pmol) into the DMH or the paraventicular nucleus (PVN) on HR, MAP, locomotor activity, and plasma ACTH in conscious rats. Chemical stimulation of the DMH with KA or BMI produced increased locomotor activity and effects on HR, MAP, and plasma ACTH that together mimicked the pattern seen in experimental stress. Similar treatment in the PVN produced only small increases in MAP. Thus activation of neurons in the region of the DMH results in increased secretion of ACTH along with other changes typically seen in experimental stress." @default.
• W2177821000 created "2016-06-24" @default.
• W2177821000 creator A5013483069 @default.
• W2177821000 creator A5023054416 @default.
• W2177821000 date "2001-01-01" @default.
• W2177821000 modified "2023-09-25" @default.
• W2177821000 title "Chemical stimulation of the dorsomedial hypothalamus elevates plasma ACTH in conscious rats" @default.
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• W2177821000 doi "https://doi.org/10.1152/ajpregu.2001.280.1.r8" @default.
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