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• W2181525098 abstract "Electrical defibrillation, the most effective treatment of life-threatening arrhythmias, such a ventricular fibrillation, is based on the application of shocks to the heart for the reversal of the abnormal rhythm. However, depending on their intensity, defibrillating shocks may cause cell injury and even death. Massive Ca 2+ influx due to electrical sarcolemmal damage contributes to both cell injury and membrane repair. This study aimed at investigating how variation in the extracellular Ca 2+ concentration ((Ca 2+ )o) affects the susceptibility of isolated cardiomyocytes to lethal injury by high-intensity electrical fields. The results indicate that doubling the physiological (Ca 2+ )o did not make the cells more vulnerable to high-intensity electric stimulation, but a 50% decrease in (Ca 2+ )o made the cells less susceptible to the shock-induced lethal injury. In order to make defibrillation procedures safer, studies have shown ways to improve defibrillators or to protect the myocardium with drugs. This study reveals a new point of view in which a safer defibrillation approach might be achieved by changing the medium around the cell." @default.
• W2181525098 created "2016-06-24" @default.
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• W2181525098 date "2014-01-01" @default.
• W2181525098 modified "2023-09-22" @default.
• W2181525098 title "INFLUENCE OF (Ca 2+ )O ON CARDIOMYOCYTE INJURY BY DEFIBRILLATOR-LIKE SHOCKS" @default.
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