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- W2181525168 abstract "In studying the pathophysiology of depressive disorders, both genetic background and environmental exposures should be considered. Elevated levels of glucocorticoids, stress hormones, caused by dysfunction of the hypothalamic-pituitary-adrenal (HPA) axis have been confirmed in both patient and animal models of depression. Such elevations disturb the beneficial functions of brain-derived neurotrophic factor (BDNF), which is essential for neuronal survival and synaptic plasticity in the central nervous system (CNS). Recently, it has been suggested that nutritional status also influences mood. Indeed, some evidence suggests possible interactions between diabetes, BDNF expression, and function of glucagon-like peptide 1 (GLP-1) which regulates insulin secretion and appetite. In this review paper, we provide current information on molecules including GLP-1 that putatively interact with BDNF signaling and respond to environmental conditions." @default.
- W2181525168 created "2016-06-24" @default.
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- W2181525168 date "2013-01-01" @default.
- W2181525168 modified "2023-09-27" @default.
- W2181525168 title "Neurotrophin Bdnf and Novel Molecular Targets in Depression" @default.
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