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- W2182589077 abstract "H. Rao, H. Hurt, M. Korczykowski, J. Giannetta, D. Shera, B. B. Avants, J. Gee, J. A. Detre, J. Wang Center for Functional Neuroimaging, Departments of Neurology and Radiology, University of Pennsylvania, Philadelphia, PA, United States, Division of Neonatology, The Children's Hospital of Philadelphia, Philadelphia, PA, United States, Division of Biostatistics & Epidemiology, The Children's Hospital of Philadelphia, Philadelphia, PA Introduction Theories and models from animal experiments have focused on interference of cocaine with corticogenesis and clearly indicated the alternations and deficits of cortical development caused by prenatal cocaine exposure (PCE), especially in the dopamine-rich areas of cerebral cortex (1). Recent studies in nonhuman primates and humans have suggested that cocaineexposed children may have altered neurobehavioral regulation patterns and the effects of PCE might not be manifested until the adulthood (2-3). However, little is known regarding the neurobiological mechanism mediating longitudinal effects of PCE on the neurocognitive development of human brain. Arterial spin labeling (ASL) perfusion MRI provides a promising means to non-invasively assess cerebral blood flow (CBF) and associated brain function, and is particularly advantageous for pediatric neuroimaging (4). In the present study, we used continuous ASL (CASL) perfusion fMRI as well as optimized voxel-based morphometry (VBM) to compare resting CBF and brain structure in prenatally cocaine exposed teenagers and socioeconomically matched controls." @default.
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- W2182589077 date "2006-01-01" @default.
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- W2182589077 title "CASL Perfusion fMRI Revealed Altered Resting Brain Function in Prenatally Cocaine Exposed Teenagers" @default.
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