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- W2182967110 abstract "The utility of herbicides is being threatened by the appearance of herbicide-resistant weeds. Resistance is evolved in weeds, when a plant survives at a dose of herbicide applied that is normally lethal to its wild type. The generals reasons for which herbicide become resistant are lack of rotation of the herbicide andherbicides with long residue period. In excluded method the site of action of weed has morphological adaptations. The second method is point mutation, results change in the gene encoding the target protein to which the herbicide normally binds site and weed biotype become resistant.In PSII herbicide molecule initiate lipid peroxidation. As a result integrity of cell membrane is lost. In plants the application of paraquat herbicide in PSIaffects the cell membrane, its integrity is lost and desiccation takes place. In ACCase resistance to herbicide have been reported due to amino acid substitution. AHAS herbicides are not competitive inhibitors within the catalytic site and mutational resistance weed do not change AHAS function. The mode of action of soil-active, pre-emergent herbicides inhibiting early cell division by binding to plant tubulin dimer.In plants, protoprphyrinogen Ix Oxidase(ppo) is a key enzyme in the biosynthesis of chlorophyll there is the loss of a 3-bp codon. Finally, itconcluded that by applications of biochemical, genetic and crop control management practices we can be able to fight our enemy of crops that is herbicide resistant weeds." @default.
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- W2182967110 date "2013-01-01" @default.
- W2182967110 modified "2023-09-24" @default.
- W2182967110 title "A Critical Review on Herbicide Resistance in Plants" @default.
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