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- W2183166999 abstract "A B S T R A C T The mechanisms involved in the production of hypoglycemic coma were studied in rabbits. Measurements were made in brain, cerebrospinal fluid (CSF), and plasma of osmolality, Na', K+, Cl1, water content, exogenous insulin, glucose, lactate, and glutamate, while pH, Pco2, Po2, and bicarbonate were evaluated in arterial blood. 35 min after i.v. injection of insulin (50 U/kg), plasma glucose did not change, but brain K+ content increased significantly. Grand mal seizures were observed in unanesthetized animals (+SD) 133±37 min after administration of insulin, at a time when brain glucose was normal, but brain tissue content of Na+, K+, osmoles, and water was significantly greater than normal. Coma supervened 212±54 min after insulin injection, at which time brain glucose, lactate, and glutamate were significantly decreased. At both 35 and 146 min after insulin administration, exogenous insulin was present in brain, but not in the CSF. After 208 min of insulin administration, animals were given i.v. glucose and sacrificed 35 min later. Most changes in the brain produced by hypoglycemia were reversed by the administration of glucose. Hypoxia (Po2=23 mm Hg) was produced and maintained for 35 min in another group of animals. Hypoxia caused" @default.
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- W2183166999 date "1974-01-01" @default.
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- W2183166999 title "EVIDENCE FOR A DIRECT EFFECT OF INSULIN ON ELECTROLYTE TRANSPORT IN BRAIN" @default.
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