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- W2184992009 abstract "La dysgenesie tubulaire renale (DTR) et l’exposition antenatale aux antagonistes du recepteur de l’angiotensine 2 (ARA2) alterent le developpement renal fœtal causant anurie et oligoamnios. Le tableau classique associe hypoplasie de la voute crânienne, sequence de Potter, hypoplasie pulmonaire, deces fœtal ou neonatal. Ce phenotype commun temoigne qu’un systeme renine-angiotensine (SRA) fœtal fonctionnel est crucial. Des mutations de genes codant pour les composants du SRA sont identifiees dans la DTR. Par ailleurs, la toxicite fœtale connue de substances interferant avec le SRA (ARA2, inhibiteurs de l’enzyme de conversion [IEC]) necessite des recommandations drastiques." @default.
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- W2184992009 date "2011-05-21" @default.
- W2184992009 modified "2023-10-18" @default.
- W2184992009 title "Altérations du système rénine-angiotensine (SRA) fœtal : entre génétique et toxicité médicamenteuse" @default.
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- W2184992009 doi "https://doi.org/10.1007/s12611-011-0103-2" @default.
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