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• W2185054482 abstract "Smoking is considered among the strongest environmental pollutants and represents a recognized high risk factor for cardiovascular diseases. Adverse cardiovascular consequences of cigarette smoking are serious and diverse as coronary heart disease, peripheral vascular disease, aortic aneurysm, myocardial infarction, cerebrovascular disease, worsened outcomes after angioplasty or bypass surgery, increased hypertension complications and sudden death. Of particular importance, there is very strong evidence that smoking actively enhances platelet activity and highly promotes platelet aggregation. This increases greatly the risk of intravascular thrombosis. At the level of coronary arteries, thus, smoking seriously amplifies the risk of coronary heart disease which may be complicated by higher mortality among middle-aged individuals. Smoking-induced platelet aggregation may be effected by several mechanisms for example enhanced inflammatory reactions, reduced monoamine oxidase activity and elevated levels of certain growth factors as platelet activating factor and thrombopoietin. Further, smoking may alter fibrin clot formation and structure encouraging intravascular clotting. On the other hand, chronic smoking may significantly impair the cardio-protective properties of certain drugs thus reduce their effectiveness in management of cardiovascular disease. Although some novel markers may be used to determine enhanced platelet activity and new therapeutic tools could be valuable in reducing adverse consequences of smoking on platelet function, yet physicians should continue to encourage and help all their smoking patients to quit." @default.
• W2185054482 created "2016-06-24" @default.
• W2185054482 creator A5005712969 @default.
• W2185054482 date "2014-01-01" @default.
• W2185054482 modified "2023-09-27" @default.
• W2185054482 title "Smoking and Platelet Function" @default.
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