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- W2185252120 abstract "Summary It has been established that there is a higher expression of neuronal isoform of nitric oxide synthase (nNOS) in the renal medulla in spontaneously hypertensive rats (SHR) compared to normotensive rats. In the present study there was investigated the role of the nitric oxide, produced by nNOS in the regulation of water and electrolyte excretion in spontaneously hypertensive rats. Experiments were carried out on conscious, male, normotensive Wistar rats and spontaneously hypertensive rats at the age of 12-14 weeks. Appropriate surgical preparation was undertaken for catheters implantation. The urine flow rate, sodium and chloride concentrations were measured. The investigated parameters did not differ between Wistar rats and SHR in control condition. The selective nNOS inhibition did not change urine flow rate in normotensive rats but in SHR nNOS inhibition provoked a decrease of urine flow rate by 50.9%. Neuronal nitric oxide synthase inhibition did not change sodium and chloride excretion neither in Wistar rats nor in SHR." @default.
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- W2185252120 date "2008-01-01" @default.
- W2185252120 modified "2023-09-27" @default.
- W2185252120 title "ROLE OF NITRIC OXIDE, PRODUCED BY nNOS IN THE REGULATION OF WATER AND ELECTROLYTE EXCRETION IN SPONTANEOUSLY HYPERTENSIVE RATS" @default.
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